"New insights into the pathogenesis of Crohn's disease: are they relevant for therapeutic options?" in Swiss Med Wkly (2009)
"Data on NOD2/CARD15 expression suggest that macrophages and epithelial cells could be the locus of the primary pathophysiological defect and that T-cell activation might just be a secondary effect inducing chronification of the inflammation, perhaps as backup mechanism to insufficient innate immunity."
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